Callisia fragrans, sometimes called the false bromeliad or false bromeliad plant, is a flowering plant species of the genus Callisia, in the spiderwort family, Commelinaceae.[1]
Callisia fragrans | |
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Scientific classification | |
Kingdom: | Plantae |
Clade: | Tracheophytes |
Clade: | Angiosperms |
Clade: | Monocots |
Clade: | Commelinids |
Order: | Commelinales |
Family: | Commelinaceae |
Genus: | Callisia |
Species: | C. fragrans |
Binomial name | |
Callisia fragrans (Lindl.) Woodson | |
Synonyms[1][2] | |
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Description
![Thumb](http://upload.wikimedia.org/wikipedia/commons/thumb/6/67/Callisia_fragrans%2C_bloeiwyse%2C_Manie_van_der_Schijff_BT%2C_a.jpg/640px-Callisia_fragrans%2C_bloeiwyse%2C_Manie_van_der_Schijff_BT%2C_a.jpg)
The fleshy stem of Callisia fragrans can grow to a height of 1 m (3.3 ft). The leaves are 25 cm (9.8 in) long and become burgundy-violet if exposed to more prolonged sunlight (an example of "sun-stressing"). Blossoms are white and fragrant.[3][4]
Range and cultivation
Callisia fragrans, also called golden tendril is endemic to Mexico, and naturalized in the West Indies, scattered locations in the United States, and a few other places.[2][5] It has been cultivated in many countries as an indoor ornamental since the early 1900s.[6] However, it can be also found growing outdoors in warmer climates in moist, fertile soil. The herb likes partially shaded areas.
Medicinal properties
It has a rich traditional reputation in Mexico as an antiviral and antimicrobial plant. In Eastern Europe, its leaves are used for the treatment of various skin diseases, burns and joint disorders.<ref name="yar"> An ethanol leaf extract (tincture) has been shown to effectively inhibit the infection of Vero cells by HSV-1, HSV-2 and an ACV-resistant strain of the latter, in vitro. However, the ethanol extract, as opposed to an aquatic extract, was ineffective against VZV.[6] Though the ethanol leaf extract had a lower selectivity index (toxicity vs. effectiveness) than ACV, it was able to inhibit the HSV-2 mutant, and may be less toxic than ACV. Direct interaction with the viruses, and the blocking of their access to l host cells, seems to be involved.[6]
References
External links
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