Argininosuccinate synthetase 1

Protein-coding gene in the species Homo sapiens From Wikipedia, the free encyclopedia

Argininosuccinate synthetase 1

Argininosuccinate synthetase is an enzyme that in humans is encoded by the ASS1 gene.[5][6][7]

Quick Facts ASS1, Available structures ...
ASS1
Available structures
PDBOrtholog search: PDBe RCSB
Identifiers
AliasesASS1, ASS, CTLN1, Argininosuccinate synthetase 1, argininosuccinate synthase 1
External IDsOMIM: 603470; MGI: 88090; HomoloGene: 6899; GeneCards: ASS1; OMA:ASS1 - orthologs
Orthologs
SpeciesHumanMouse
Entrez
Ensembl
UniProt
RefSeq (mRNA)

NM_000050
NM_054012

NM_007494

RefSeq (protein)

NP_000041
NP_446464

NP_031520

Location (UCSC)Chr 9: 130.44 – 130.5 MbChr 2: 31.36 – 31.41 Mb
PubMed search[3][4]
Wikidata
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The protein encoded by this gene catalyzes the penultimate step of the arginine biosynthetic pathway. There are approximately 10 to 14 copies of this gene including the pseudogenes scattered across the human genome, among which the one located on chromosome 9 appears to be the only functional gene for argininosuccinate synthetase. Two transcript variants encoding the same protein have been found for this gene.[7]

Clinical significance

Mutations in the chromosome 9 copy of ASS cause citrullinemia.[5]

Arginine is considered a non-essential amino acid since normal cells can synthesize it from citrulline and aspartate using the enzymes argininosuccinate synthase 1 (ASS1) and argininosuccinate lyase (ASL). Consequently, depleting arginine can be an effective therapeutic approach. Notably, over 70% of tumors show reduced ASS1 transcription, making these cancer cells reliant on external sources of arginine, which forms the basis of arginine-deprivation therapy.[8]

The investigational drug pegargiminase that degrades arginine is currently in trials for the treatment of ASS1 deficient cancers.[9]

References

Further reading

Further reading

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